Hua Ni | Biotechnology | Best Researcher Award
Mr. Hua Ni | Kashi University | China
Dr. Hua Ni is a Lecturer at the School of Life and Geographic Sciences, Kashgar University, specializing in cell biology and molecular mechanisms of disease. She earned her Ph.D. in Cell Biology from Nankai University (2020–2024), following an M.Sc. in Cell Biology from Nanjing Normal University (2013–2016) and a B.Sc. in Biotechnology from Shihezi University (2009–2013). Since joining Kashgar University in 2016 and advancing to Lecturer in 2019, Dr. Ni has been actively engaged in both teaching and research. Her research portfolio demonstrates a strong focus on cellular regulation, infectious diseases, and tissue homeostasis. She has contributed as a participant to major National Natural Science Foundation of China (NSFC) projects, including studies on the cannabinoid signaling system in sheep intramuscular fat formation (2026–2029) and on the molecular regulation of Streptococcus suis virulence (2021–2024). In addition, she serves as Principal Investigator of a Xinjiang Uygur Autonomous Region Youth Fund project exploring the role of glycocalyx in inflammatory bowel disease (2026–2028). Dr. Ni has published several impactful papers in leading international journals, with notable contributions such as uncovering the role of O-GlcNAcylation in regulating phagocytosis (Journal of Genetics and Genomics, 2023), demonstrating the pathogenic effects of htpsA gene inactivation in Streptococcus suis (Virulence, 2020), and identifying dynamic ciliary changes essential for corneal homeostasis (Journal of Cellular Physiology, 2024). She has also advanced understanding of pulmonary fibrosis by revealing CYLD/HDAC6 signaling pathways that regulate epithelial-mesenchymal transition and ciliary homeostasis (Cell Death & Disease, 2024). Recognized as both sole first author and co-first author on several publications, Dr. Ni’s research highlights her growing influence in cell biology and disease mechanisms. Her academic journey reflects consistent dedication to advancing biomedical science, contributing to regional research development, and preparing the next generation of life scientists through her teaching and mentorship.
Profile: Orcid
Featured Publications
Ni, H., Chen, M., Dong, D., Zhou, Y., Cao, Y., Ge, R., Luo, X., Wang, Y., Dong, X., Zhou, J., et al. (2024). CYLD/HDAC6 signaling regulates the interplay between epithelial–mesenchymal transition and ciliary homeostasis during pulmonary fibrosis. Cell Death & Disease, 15, 69572.
Ni, H., Li, L., Hu, D., Yang, M., Wang, D., Ma, H., Bu, W., Yang, J., Zhu, L.-E., Zhai, D., et al. (2024). Dynamic changes of endothelial and stromal cilia are required for the maintenance of corneal homeostasis. Journal of Cellular Physiology, 239(5), 31215.
Yu, F., Yang, S., Ni, H., Heng, D., Wu, X., Yang, M., Zhang, X., Cao, Y., Pei, Y., An, D., et al. (2023). O-GlcNAcylation regulates centrosome behavior and cell polarity to reduce pulmonary fibrosis and maintain the epithelial phenotype. Advanced Science, 10(12), 3545.
Ni, H., Li, M., Wang, Q., Wang, J., Liu, X., Zheng, F., Hu, D., Yu, X., Han, Y., Zhang, Q., et al. (2020). Inactivation of the htpsA gene affects capsule development and pathogenicity of Streptococcus suis. Virulence, 11(1), 1792080.
Ni, H., Fan, W., Li, C., Wu, Q., Hou, H., Hu, D., Zheng, F., Zhu, X., Wang, C., Cao, X., et al. (2018). Streptococcus suis DivIVA protein is a substrate of Ser/Thr kinase STK and involved in cell division regulation. Frontiers in Cellular and Infection Microbiology, 8, 85.